vasoactive intestinal peptide Search Results


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Elabscience Biotechnology mouse vip elisa kit
FIGURE 3 Vasoactive intestinal peptide <t>(VIP)</t> upregulated PTEN, VIP receptor mRNA expression, and Treg‐related cytokines in spleen. Q‐PCR results for VIP mRNA, VIP receptors mRNA expression (A), and PTEN/PI3K/AKT pathway (B) in the spleen. The concentrations of VIP in the serum (C) were measured using <t>ELISA</t> kits (n = 7). The abundance of interleukin (IL)‐10 (D) and IL‐2 (E) in the splenic tissue homogenate supernatant was determined by ELISA as well (n = 7). Unpaired Student t‐test was used in the analysis. *p < 0.05, **p < 0.01, ***p < 0.001.
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Bio-Rad rabbit anti vasoactive intestinal peptide
FIGURE 3 Vasoactive intestinal peptide <t>(VIP)</t> upregulated PTEN, VIP receptor mRNA expression, and Treg‐related cytokines in spleen. Q‐PCR results for VIP mRNA, VIP receptors mRNA expression (A), and PTEN/PI3K/AKT pathway (B) in the spleen. The concentrations of VIP in the serum (C) were measured using <t>ELISA</t> kits (n = 7). The abundance of interleukin (IL)‐10 (D) and IL‐2 (E) in the splenic tissue homogenate supernatant was determined by ELISA as well (n = 7). Unpaired Student t‐test was used in the analysis. *p < 0.05, **p < 0.01, ***p < 0.001.
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Cusabio mouse vasoactive intestinal peptide vip elisa kit
FIGURE 3 Vasoactive intestinal peptide <t>(VIP)</t> upregulated PTEN, VIP receptor mRNA expression, and Treg‐related cytokines in spleen. Q‐PCR results for VIP mRNA, VIP receptors mRNA expression (A), and PTEN/PI3K/AKT pathway (B) in the spleen. The concentrations of VIP in the serum (C) were measured using <t>ELISA</t> kits (n = 7). The abundance of interleukin (IL)‐10 (D) and IL‐2 (E) in the splenic tissue homogenate supernatant was determined by ELISA as well (n = 7). Unpaired Student t‐test was used in the analysis. *p < 0.05, **p < 0.01, ***p < 0.001.
Mouse Vasoactive Intestinal Peptide Vip Elisa Kit, supplied by Cusabio, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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FIGURE 3 Vasoactive intestinal peptide <t>(VIP)</t> upregulated PTEN, VIP receptor mRNA expression, and Treg‐related cytokines in spleen. Q‐PCR results for VIP mRNA, VIP receptors mRNA expression (A), and PTEN/PI3K/AKT pathway (B) in the spleen. The concentrations of VIP in the serum (C) were measured using <t>ELISA</t> kits (n = 7). The abundance of interleukin (IL)‐10 (D) and IL‐2 (E) in the splenic tissue homogenate supernatant was determined by ELISA as well (n = 7). Unpaired Student t‐test was used in the analysis. *p < 0.05, **p < 0.01, ***p < 0.001.
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Cusabio elisa kit
FIGURE 3 Vasoactive intestinal peptide <t>(VIP)</t> upregulated PTEN, VIP receptor mRNA expression, and Treg‐related cytokines in spleen. Q‐PCR results for VIP mRNA, VIP receptors mRNA expression (A), and PTEN/PI3K/AKT pathway (B) in the spleen. The concentrations of VIP in the serum (C) were measured using <t>ELISA</t> kits (n = 7). The abundance of interleukin (IL)‐10 (D) and IL‐2 (E) in the splenic tissue homogenate supernatant was determined by ELISA as well (n = 7). Unpaired Student t‐test was used in the analysis. *p < 0.05, **p < 0.01, ***p < 0.001.
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FIGURE 3 Vasoactive intestinal peptide <t>(VIP)</t> upregulated PTEN, VIP receptor mRNA expression, and Treg‐related cytokines in spleen. Q‐PCR results for VIP mRNA, VIP receptors mRNA expression (A), and PTEN/PI3K/AKT pathway (B) in the spleen. The concentrations of VIP in the serum (C) were measured using <t>ELISA</t> kits (n = 7). The abundance of interleukin (IL)‐10 (D) and IL‐2 (E) in the splenic tissue homogenate supernatant was determined by ELISA as well (n = 7). Unpaired Student t‐test was used in the analysis. *p < 0.05, **p < 0.01, ***p < 0.001.
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FIGURE 3 Vasoactive intestinal peptide <t>(VIP)</t> upregulated PTEN, VIP receptor mRNA expression, and Treg‐related cytokines in spleen. Q‐PCR results for VIP mRNA, VIP receptors mRNA expression (A), and PTEN/PI3K/AKT pathway (B) in the spleen. The concentrations of VIP in the serum (C) were measured using <t>ELISA</t> kits (n = 7). The abundance of interleukin (IL)‐10 (D) and IL‐2 (E) in the splenic tissue homogenate supernatant was determined by ELISA as well (n = 7). Unpaired Student t‐test was used in the analysis. *p < 0.05, **p < 0.01, ***p < 0.001.
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Cusabio rat vasoative intestinal peptide vip enzyme linked immunosorbent assay elisa kit
FIGURE 3 Vasoactive intestinal peptide <t>(VIP)</t> upregulated PTEN, VIP receptor mRNA expression, and Treg‐related cytokines in spleen. Q‐PCR results for VIP mRNA, VIP receptors mRNA expression (A), and PTEN/PI3K/AKT pathway (B) in the spleen. The concentrations of VIP in the serum (C) were measured using <t>ELISA</t> kits (n = 7). The abundance of interleukin (IL)‐10 (D) and IL‐2 (E) in the splenic tissue homogenate supernatant was determined by ELISA as well (n = 7). Unpaired Student t‐test was used in the analysis. *p < 0.05, **p < 0.01, ***p < 0.001.
Rat Vasoative Intestinal Peptide Vip Enzyme Linked Immunosorbent Assay Elisa Kit, supplied by Cusabio, used in various techniques. Bioz Stars score: 85/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Tocris p5 mouse brains
A–D, Effects of CUMS on excitotoxic brain lesions induced by ibotenate (A–D), NMDA (E, F), <t>or</t> <t>S-bromowillardiine</t> (G, H) in C57BL/6 mice. Excitotoxins were injected at <t>P5,</t> and pups were killed at P10. Bars represent mean sagittal diameter (A, C, E, G) or mean volume (B, D, F, H) of the lesions ± SEM. Asterisks indicate statistically significant difference from control males (A–D) or controls (E–H). *p < 0.05, **p < 0.01, ***p < 0.001 in three-way ANOVA with contrasts (A–G) or in Student's t test (E–H). Ctrl, Control.
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Image Search Results


FIGURE 3 Vasoactive intestinal peptide (VIP) upregulated PTEN, VIP receptor mRNA expression, and Treg‐related cytokines in spleen. Q‐PCR results for VIP mRNA, VIP receptors mRNA expression (A), and PTEN/PI3K/AKT pathway (B) in the spleen. The concentrations of VIP in the serum (C) were measured using ELISA kits (n = 7). The abundance of interleukin (IL)‐10 (D) and IL‐2 (E) in the splenic tissue homogenate supernatant was determined by ELISA as well (n = 7). Unpaired Student t‐test was used in the analysis. *p < 0.05, **p < 0.01, ***p < 0.001.

Journal: Immunity, inflammation and disease

Article Title: Vasoactive intestinal peptide exerts therapeutic action by regulating PTEN in a model of Sjögren's disease.

doi: 10.1002/iid3.936

Figure Lengend Snippet: FIGURE 3 Vasoactive intestinal peptide (VIP) upregulated PTEN, VIP receptor mRNA expression, and Treg‐related cytokines in spleen. Q‐PCR results for VIP mRNA, VIP receptors mRNA expression (A), and PTEN/PI3K/AKT pathway (B) in the spleen. The concentrations of VIP in the serum (C) were measured using ELISA kits (n = 7). The abundance of interleukin (IL)‐10 (D) and IL‐2 (E) in the splenic tissue homogenate supernatant was determined by ELISA as well (n = 7). Unpaired Student t‐test was used in the analysis. *p < 0.05, **p < 0.01, ***p < 0.001.

Article Snippet: Serum extraction method as published,20 VIP levels in serum were measured using the mouse VIP ELISA kit (Elabscience).

Techniques: Expressing, Enzyme-linked Immunosorbent Assay

A–D, Effects of CUMS on excitotoxic brain lesions induced by ibotenate (A–D), NMDA (E, F), or S-bromowillardiine (G, H) in C57BL/6 mice. Excitotoxins were injected at P5, and pups were killed at P10. Bars represent mean sagittal diameter (A, C, E, G) or mean volume (B, D, F, H) of the lesions ± SEM. Asterisks indicate statistically significant difference from control males (A–D) or controls (E–H). *p < 0.05, **p < 0.01, ***p < 0.001 in three-way ANOVA with contrasts (A–G) or in Student's t test (E–H). Ctrl, Control.

Journal: The Journal of Neuroscience

Article Title: Chronic Mild Stress during Gestation Worsens Neonatal Brain Lesions in Mice

doi: 10.1523/JNEUROSCI.5330-06.2007

Figure Lengend Snippet: A–D, Effects of CUMS on excitotoxic brain lesions induced by ibotenate (A–D), NMDA (E, F), or S-bromowillardiine (G, H) in C57BL/6 mice. Excitotoxins were injected at P5, and pups were killed at P10. Bars represent mean sagittal diameter (A, C, E, G) or mean volume (B, D, F, H) of the lesions ± SEM. Asterisks indicate statistically significant difference from control males (A–D) or controls (E–H). *p < 0.05, **p < 0.01, ***p < 0.001 in three-way ANOVA with contrasts (A–G) or in Student's t test (E–H). Ctrl, Control.

Article Snippet: Excitotoxic lesions and lesion size determination Excitotoxic brain lesions were induced by injecting 10 μg of ibotenate (NMDA and metabotropic receptor agonist; Tocris Bioscience, Bristol, UK), 5 μg of NMDA (NMDA receptor agonist; Tocris Bioscience), or 15 μg of S-bromowillardiine (AMPA and kainate receptor agonist; Tocris Bioscience) into P5 mouse brains.

Techniques: Injection

Real-time PCR quantification of glucocorticoid receptor (GR) mRNA. Pups from control (Ctrl) and GR-I unstressed B6C3F1 mothers were killed at P0 or P5. Brains were removed, and cortical plate (A) and underlying white matter (B) immediately adjacent to the normal site of excitotoxin injection were collected for RNA extraction. Bars represent mean GR/β2-microglobulin ratios ± SEM. Asterisks indicate statistically significant differences between Ctrl pups from unstressed mothers and pups from mothers exposed to CUMS. *p < 0.05, **p < 0.01, ***p < 0.001 in ANOVA with Dunnett's test.

Journal: The Journal of Neuroscience

Article Title: Chronic Mild Stress during Gestation Worsens Neonatal Brain Lesions in Mice

doi: 10.1523/JNEUROSCI.5330-06.2007

Figure Lengend Snippet: Real-time PCR quantification of glucocorticoid receptor (GR) mRNA. Pups from control (Ctrl) and GR-I unstressed B6C3F1 mothers were killed at P0 or P5. Brains were removed, and cortical plate (A) and underlying white matter (B) immediately adjacent to the normal site of excitotoxin injection were collected for RNA extraction. Bars represent mean GR/β2-microglobulin ratios ± SEM. Asterisks indicate statistically significant differences between Ctrl pups from unstressed mothers and pups from mothers exposed to CUMS. *p < 0.05, **p < 0.01, ***p < 0.001 in ANOVA with Dunnett's test.

Article Snippet: Excitotoxic lesions and lesion size determination Excitotoxic brain lesions were induced by injecting 10 μg of ibotenate (NMDA and metabotropic receptor agonist; Tocris Bioscience, Bristol, UK), 5 μg of NMDA (NMDA receptor agonist; Tocris Bioscience), or 15 μg of S-bromowillardiine (AMPA and kainate receptor agonist; Tocris Bioscience) into P5 mouse brains.

Techniques: Real-time Polymerase Chain Reaction, Injection, RNA Extraction